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Shigeru Tanaka serves as Associate Professor in the Graduate School of Biomedical and Health Sciences at Hiroshima University. He earned a Doctor of Philosophy in Medical Science from Osaka University. His research focuses on molecular and pharmacological neuroscience, with particular emphasis on G protein-coupled receptors such as GPR3, protein kinase C signaling, neuronal protection mechanisms, autophagy, stroke, and related pathways in neurological function and disease. Tanaka teaches undergraduate and graduate courses including Biological Responses, Medical Neuroscience II, and Practice for medical research.
Tanaka has authored numerous peer-reviewed publications. Key recent works include studies on protein kinase C inhibitor Calphostin C published in the European Journal of Pharmacology in 2024, S-palmitoylation of the serotonin transporter in Biochemical and Biophysical Research Communications in 2023, features of propofol-induced protein kinase C translocation in Frontiers in Pharmacology in 2023, and the role of P2Y2 receptor in dying cell removal in Journal of Pharmacological Sciences in 2023. Earlier contributions address GPR3 expression in retinal ganglion cells in Neurobiology of Disease in 2022, GPR3 in neurite outgrowth in Molecular and Cellular Neuroscience in 2022, and developmental expression of GPR3 in Neurobiology of Disease in 2014. Additional papers examine propofol effects on calcium signaling, histone deacetylase 10 in autophagy, serotonin transporter regulation, and tau protein modifications after cerebral ischemia. His affiliations include the Society for Neuroscience, the Japan Academy of Neurosonology, and the Molecular Biology Society of Japan.
Hiroshima University researchers reveal how GPR3 acts like an immediate-early gene to influence early neuron development, with implications for brain plasticity and disorders.
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