Helps students build confidence and skills.
Encourages students to think critically.
Always respectful and encouraging to all.
Passionate about student development.
Helps students see the value in learning.
Dr Emma Hadfield serves as a Lecturer in the Curtin School of Diagnostic and Therapeutic Sciences within the Faculty of Health Sciences at Curtin University. She is affiliated with Curtin Medical School and plays a key role in delivering education in clinical biochemistry. Her teaching responsibilities include units such as BCCB2000 Foundations of Biochemistry in Semester 1 2024, MEDI2004 Foundations of Clinical Biochemistry in Semester 2 2020, and MEDI4003 Advanced Clinical Biochemistry in Semester 2 2022. These courses cover foundational and advanced topics in biochemistry relevant to diagnostic and therapeutic sciences.
Hadfield earned her Doctor of Philosophy (PhD) from The University of Western Australia, with research focused on lipoprotein oxidation. Prior to her current academic appointment, she gained experience in the publishing industry, complementing her transition into higher education teaching and research. Her academic interests center on oxidative stress and its implications in neurodegenerative diseases, particularly Alzheimer's disease. Under her previous name, Waddington E., she contributed to significant publications in this area. These include the book chapter 'Beta-amyloid and oxidative stress in the pathogenesis of Alzheimer's Disease' (1999, with Veurink G, Laws S, Croft K, Dharmarajan AM) and the journal article 'Plasma F2-isoprostane levels are increased in Alzheimer's disease: evidence of increased oxidative stress in vivo' (with Croft K, Clarnette R, Mori T, Martins R). Additional works encompass 'Fatty Acid Oxidation in the Pathogenesis of Alzheimer's Disease' and contributions to reviews on genetics, lifestyle, amyloid beta, and oxidative stress in Alzheimer's disease. These publications highlight her involvement in exploring biomarkers like F2-isoprostanes and the role of oxidative injury in Alzheimer's etiology, providing evidence for elevated oxidative stress in affected patients. Her research aligns with broader investigations into beta-amyloid aggregation and neurotoxicity mechanisms.
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